publication list

Recent Publications:

Fogelson, B.G., Nawas, S.I., and LAW, W.R. Mechanisms of Myocardial Protection in Adenosine-Supplemented Cardioplegia: Myofilament and Metabolic Responses. J. Thorac. Cardiovasc. Surg. 119: 601-609, 2000
Sam, A.D., II, Sharma, A.C., Rice, A.N., Ferguson, J.L., and LAW, W.R. Adenosine and Nitric Oxide Regulate Regional Vascular Resistance via Interdependent and Independent Mechanisms during Sepsis. Crit. Care Med. 28:1931-1939, 2000.
Adanin, S.,Yalovetskiy, I.V., Narduli, B.A., Sam, A.D., II, Jonjev, Z.S. and LAW, W.R. Inhibiting Adenosine Deaminase Modulates the Systemic Inflammatory Response Syndrome in Endotoxemia and Sepsis, American Journal of Physiology 282: R1324-R1332, 2002.
Cohen, E.S, LAW, W.R., Easington, C.R., Cruz, K.Q., Nardulli, B.A., Balk, R.A., Parrillo, J.E., and Hollenberg, S.M. Adenosine Deaminase Inhibition Attenuates Microvascular Dysfunction and Improves Survival in Sepsis. Am. J. Respir. Crit. Care Med. 166:16-20, 2002.
LAW, W.R., Valli, V.E., and Conlon, B.A. Therapeutic Potential for Transient Inhibition of Adenosine Deaminase in Systemic Inflammatory Response Syndrome. Critical Care Medicine 31:1475-1481, 2003.
Jonjev, Z.S., Schwertz, D.W., Beck, J.M., Ross, J.D., and LAW, W.R. Subcellular distribution of protein kinase C isozymes during cardioplegic arrest. J. Thoracic and Cardiovascular Surgery. 126:1880-1885, 2003.
Conlon, B.A. and LAW, W.R. Macrophages Are a Source of Extracellular Adenosine Deaminase-2 During Inflammatory Responses. Clin. Exper. Immun. (In Press), 2004
LAW, W.R., Ross, J.D., and Jonjev, Z.S. Adenosine attenuates C-terminal but not N-terminal proteolysis of cTnI during cardioplegic arrest. J. Surg. Res. (In Press) 2004

Recent Publications:

Fogelson, B.G., Nawas, S.I., and LAW, W.R. Mechanisms of Myocardial Protection in Adenosine-Supplemented Cardioplegia: Myofilament and Metabolic Responses. J. Thorac. Cardiovasc. Surg. 119: 601-609, 2000

Sam, A.D., II, Sharma, A.C., Rice, A.N., Ferguson, J.L., and LAW, W.R. Adenosine and Nitric Oxide Regulate Regional Vascular Resistance via Interdependent and Independent Mechanisms during Sepsis. Crit. Care Med. 28:1931-1939, 2000.

Adanin, S.,Yalovetskiy, I.V., Narduli, B.A., Sam, A.D., II, Jonjev, Z.S. and LAW, W.R. Inhibiting Adenosine Deaminase Modulates the Systemic Inflammatory Response Syndrome in Endotoxemia and Sepsis, American Journal of Physiology 282: R1324-R1332, 2002.

Cohen, E.S, LAW, W.R., Easington, C.R., Cruz, K.Q., Nardulli, B.A., Balk, R.A., Parrillo, J.E., and Hollenberg, S.M. Adenosine Deaminase Inhibition Attenuates Microvascular Dysfunction and Improves Survival in Sepsis. Am. J. Respir. Crit. Care Med. 166:16-20, 2002.

LAW, W.R., Valli, V.E., and Conlon, B.A. Therapeutic Potential for Transient Inhibition of Adenosine Deaminase in Systemic Inflammatory Response Syndrome. Critical Care Medicine 31:1475-1481, 2003.

Jonjev, Z.S., Schwertz, D.W., Beck, J.M., Ross, J.D., and LAW, W.R. Subcellular distribution of protein kinase C isozymes during cardioplegic arrest. J. Thoracic and Cardiovascular Surgery. 126:1880-1885, 2003.

Conlon, B.A. and LAW, W.R. Macrophages Are a Source of Extracellular Adenosine Deaminase-2 During Inflammatory Responses. Clin. Exper. Immun. (In Press), 2004

LAW, W.R., Ross, J.D., and Jonjev, Z.S. Adenosine attenuates C-terminal but not N-terminal proteolysis of cTnI during cardioplegic arrest. J. Surg. Res. (In Press) 2004